After recovery they can develop post-polio syndrome years later as adults, causing weakness or paralysis. The CNS, by virtue of its dependence on a high and uninterrupted level of energy production, is likely to be significantly affected by energy deprivation. It affects all ages and both sexes, sometimes as an epidemic in feedlot cattle or sheep. In the less severe cases, cattle with polio show head-pressing and remain stationary for several hours or a few days and will show anorexia. Treatment- HIGH doses of Pen G every 6 hours until all symptoms has disappeared. Content uploaded by Fabiano Sant'Ana. The first sign of a problem is generally cattle exhibiting polio symptoms. Suspected based on clinical signs and response to thiamine; confirmed by necropsy. The animal (usually a 400- to 600-lb. A neurologic disorder diagnosed in Australia has been associated with the Nardoo fern (Marsilea drummondii), which may contain high levels of a thiaminase I enzyme. A common name for this disease in sheep and goats is “polio”; however, it has absolutely no relationship with the infectious viral disease found in humans (poliomyelitis). Gross lesions due to polioencephalomalacia are inconsistent and frequently subtle, especially early in the disease. It briefly discusses clinical signs, necropsy findings, causes, and a Bovine Diet. Although PEM has been produced experimentally by feeding high doses of extracts of such plants, field cases are uncommon, because these plants are unpalatable. Water consumption by cattle is temperature dependent and increases greatly at high temperatures, leading to increased sulfur intake due to concurrent increases in water consumption and sulfate concentrations in water. Usually, affected sheep or cattle display signs for 1–6 days before dying. Beneficial effects are usually seen within 24 hours and sometimes sooner; however, if there is no initial improvement, treatment should be continued for ≥3 days. I say this because I had wondered that same thing, still being on the fence w vaccines, it seemed that polio existed before, after and outside of DDT. When cattle undergo a transition to high sulfur intake, ruminal sulfide concentrations peak 1–4 weeks after the change. Most commonly, decreased thiamine concentration in the diet or high levels of dietary sulfates are the cause of polio in growing and feedlot cattle. In the 1940s, Frederick R. Klenner, MD, had 100% success rate treating polio-like symptoms with intravenous vitamin C. At the same time, in Australia, Sister Kenney successfully used physical therapy with polio victims ... Cattle in Australia were routinely dipped in chemicals, including arsenic. Maybe there are more ways to cause polio symptoms, like there are several ways to achieve a headache, maybe DDT was just one of the many ways a person developed polio like symptoms. Younger animals are more frequently affected than adults. Death occurs rapidly if cattle are not treated. Polio is a contagious viral illness that in its most severe form causes nerve injury leading to paralysis, difficulty breathing and sometimes death.In the U.S., the last case of naturally occurring polio was in 1979. True polioencephalomalacia, on the other hand, has a known cause. Treatment consists of intravenous injection of thiamin and glucose. Cases of PEM can be successfully treated if detected early in the disease course, making recognition of early symptoms a critical issue for sheep and goat producers. Polio in Cattle can be Caused by Sulfur Toxicity. During a PEM outbreak, sufficient roughage should be provided. This causes rapid problems for the animal. Preruminant animals depend on dietary thiamine. It is also known as abortive polio. The initial histologic lesions are necrosis of cerebrocortical neurons. Polio is a serious viral infection that used to be common in the UK and worldwide. 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